Strategic Plan Question Detail
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Question 2: How Can I Understand What Is Happening? (Biology)  

$63,252,948.71
Fiscal Year: 2009

Download 2009 Question 2 projects (EXCEL)
Note: Initial Sort is by Principal Investigator. Sorting by other columns is available by clicking on the desired column header.
Project Title Principal Investigator Strategic Plan Objective Institution
CD8 + T lymphocyte function in autism Ashwood, Paul Q2.S.A University of California, Davis
CD8 + T lymphocyte function in autism Ashwood, Paul Q2.S.A University of California, Davis
Primate models of autism Bauman, Melissa Q2.S.A University of California, Davis
Maternal inflammation alters fetal brain development via tumor necrosis factor-alpha Carpentier, Pamela Q2.S.A Stanford University
Modulation of neuronal cysteine uptake and redox status by morphine, gluten/casein-derived opiates and naltrexone Deth, Richard Q2.S.A Northeastern University School of Pharmacy
The pathogenesis of autism: Maternal antibody exposure in the fetal brain Diamond, Betty Q2.S.A The Feinstein Institute for Medical Research
Relationship between celiac disease and autism Russo, A.J. Q2.S.A Health Research Institute
Mechanisms of mitochondrial dysfunction in autism Shoffner, John Q2.S.A Georgia State University
Neuroimmunologic investigations of autism spectrum disorders (ASD) Swedo, Susan Q2.S.A National Institutes of Health (NIH)
Project 2: Immunological susceptibility of autism Van de Water, Judy Q2.S.A University of California, Davis
A mitochondrial etiology of autism Wallace, Douglas Q2.S.A University of California, Irvine
Autism: Role of oxytocin Ellerbeck, Kathryn Q2.S.A University of Kansas Medical Center
A role for immune molecules in cortical connectivity: Potential implications for autism Elmer, Bradford Q2.S.A University of California, Davis
Is autism a mitochondrial disease? Giulivi, Cecilia Q2.S.A University of California, Davis
Redox abnormalities as a vulnerability phenotype for autism and related alterations in CNS development Hepel, Maria Q2.S.A State University of New York at Potsdam
How does IL-6 mediate the development of autism-related behaviors? Hsiao, Elaine Q2.S.A California Institute of Technology
Redox abnormalities as a vulnerability phenotype for autism and related alterations in CNS development James, Sandra Q2.S.A Arkansas Children's Hospital Research Institute
Impact of innate immunity on T and B cell differentiation in autistic children/Altered TLR response in a subset of children with regressive autism Jyonouchi, Harumi Q2.S.A University of Medicine & Dentistry of New Jersey
Impact of innate immunity on T and B cell differentiation in autistic children/Altered TLR response in a subset of children with regressive autism Jyonouchi, Harumi Q2.S.A University of Medicine & Dentistry of New Jersey
Impact of innate immunity on regressive autism Jyonouchi, Harumi Q2.S.A University of Medicine & Dentistry of New Jersey
Repository for tissues from children with and without autism Kushak, Rafail Q2.S.A Massachusetts General Hospital
Regulation of inflammatory Th17 cells in autism spectrum disorder Littman, Dan Q2.S.A New York University School of Medicine
Immune molecules and cortical synaptogenesis: Possible implications for the pathogenesis of autism McAllister, A. Kimberley Q2.S.A University of California, Davis
Influence of oxidative stress on transcription and alternative splicing of methionine synthase in autism Muratore, Christina Q2.S.A Northeastern University
Redox abnormalities as a vulnerability phenotype for autism and related alterations in CNS development Noble, Mark Q2.S.A University of Rochester